#845 BIO CELL HomoCysteine TLR
HomoCysteine TLR #845 Category: BIO CELL
A comprehensive formula and next generation of natural support for the body’s innate homocysteine methylation processes innovated by Dr. Shayne Morris.
This product provides essential homocysteine nutrients and herbalomic ingredients for homocysteine-thiolactone, a toxin that attacks many types of proteins, especially in the blood and brain.
The key benefits of this formula:
efficiently deplete plasma homocysteine levels
reduce the dangerous metabolic chemicals of heightened homocysteine and homocysteine thiolactone
increase the required nutrition to decrease oxidative stress, hydrogen peroxide, and organophosphate pesticides and glyphosate
increase the PON1 enzyme in the liver and kidneys, which in turn detoxifies homocysteine thiolactone and protects the body from LDL cholesterol oxidation
antioxidants that protect the heart, cardiovascular system, and the brain from the dangerous metabolic chemicals of elevated homocysteine
provide key micronutrients required to convert homocysteine to glutathione
provide 3 forms of vitamin B12, essential for homocysteine metabolic pathways.
What is homocysteine and why is it important to be aware of?
Homocysteine is a naturally occuring amino acid produced as part of the body’s methylation process and as a result of breaking down proteins.
Elevated homocysteine levels in the plasma, also known as hyperhomocysteinemia, can negatively impact the heart, cardiovascular functions, and the brain. Elevated levels are dangerous metabolic chemicals that bind with tissues and cause oxidative stress. Futhermore, elevated homocysteine levels are commonly linked to a deficiency in both vitamin B12 and folate.
Heightened homocysteine levels can have a dramatic impact on our health, affecting systems like methylation, hormones, stress and anxiety, immune function and cardiovascular performance.
Rest assured, Homocysteine TLR provides a synergistic blend of precursor nutrients that increase the PON1 enzyme, which in turn decreases elevated homocysteine levels.
The PON1 enzyme is a high density lipoprotein (HDL) enzyme that is capable of protecting against lipid oxidation, decreasing oxidative stress, organophosphate pesticides and glyphosate.
There are a plethora of key micronutrients and compounds in the Homocysteine TLR formula that help with homocysteine regulation, help to convert homocysteine to glutathione and help with homocysteine metabolic processes. Furthermore, there are antioxidants that protect the heart, the cardiovascular system, and the brain.
What is considered high or low
• High homocysteine is like high glucose: you need some, but too much is
toxic and too little results in metabolic dysfunction.
• Current lab ranges is usually 4-15(μmol/L) before homocysteine is
considered elevated. Functional lab range is 6.0-7.5(μmol/L).
• Above 7.5 is considered functionally elevated. When homocysteine lab
test first came out the range was up to 7.5, but then was adjusted to be
higher to fit a bell curve.
• High homocysteine is a marker of metabolic dysfunction, inflammation,
tissue damage, especially methylation disruption.
• Low homocysteine may indicate oxidative stress leading to homocysteine
being converted into glutathione.
• Many doctors are not testing homocysteine, or even considering
elevated homocysteine to be a significant health risk.
• Measuring homocysteine is an inexpensive, easy, and highly informative
tool that indirectly measures inflammation, B-vitamin levels, and overall
health of the body.
• No readily available lab test for measuring homocysteine-thiolactone, so
depends greatly on homocysteine and genetic testing.
*Diseases associated with high homocysteine
• Alzheimer’s disease
• Birth defects
• Blood clots
• Coronary artery disease
• Endothelial damage, which leads to cardiovascular disease
• Parkinson's disease
• Vascular disease
• Leaky Blood brain barrier leading to neuroinflammation.
• Down syndrome
• Diabetic retinopathy
• Macular Degernation
• Cognitive impairment.
• Subcortical damage
• Brain atrophy
• Decreased cerebral eNOS activity.
• Decreased glucose transporter-1
• Increased VCAM-1 (Vascular cell adhesion protein 1 )
• Increased arterial stiffness in prehypertensive patients.
• Digestive diseases: including constipation, inflammatory
bowel disease, Crohn’s disease, and colorectal cancer.
• Homocysteine can cause programmed cell death in human
vascular endothelial cells by interfering with protein
• Smaller Brain Volume
• Decreased hippocampal volume.